No it's different. The article is a bit misleading because it's not really just a pharmacological tourniquet, rather it prevents some of the secondary effects of trauma - probably what is killing the patients with the 'butterfly' effect.
The article doesn't mention it, but all the studies done with it include a 7.5% hypertonic saline solution which restores circulatory volume. The adenosine, lidocaine and magnesium decreases some of the nasty mediators (eg TNF) that severe trauma stimulates. It's also been tried post cardiac arrest and with animals shocked with septicaemia.. The abstract below, from one of Prof. Dunbars articles is a good summary of it's effects, the last line is very important.
Adenosine, lidocaine, and Mg2+ (ALM): From cardiac surgery to combat casualty care--Teaching old drugs new tricks.
New frontline drugs and therapies are urgently required to protect the body from primary and secondary injuries. We review more than 10 years of work on adenosine, lidocaine, and magnesium (ALM) and its possible significance to civilian and military medicine. Adenosine is an endogenous nucleoside involved in nucleotide production, adenosine triphosphate turnover, and restoration of supply and demand imbalances. Lidocaine is a local anaesthetic and Class 1B antiarrhythmic, and magnesium is essential for ionic regulation and cellular bioenergetics. Individually, each plays important roles in metabolism, immunomodulation, inflammation, and coagulation. The original idea to combine all three was as a "polarizing" cardioplegia, an idea borrowed from natural hibernators. Two recent prospective, randomized human trials have demonstrated its safety and superiority in myocardial protection over high-potassium "depolarizing" solutions. The next idea came from witnessing how the human heart spontaneously reanimated after complex operations with little inotropic support. At high doses, ALM arrests the heart, and at lower doses, it resuscitates the heart. In rat and pig models, we have shown that ALM intravenous bolus and infusion "drip" protects against acute regional myocardial ischemia, lethal arrhythmias, cardiac arrest, compressible and noncompressible blood loss and shock, endotoxemia, and sepsis. Individually, adenosine, lidocaine, or magnesium fails to protect. Protection is afforded in part by reducing inflammation, correcting coagulopathy, and lowering energy demand. We propose a unifying hypothesis involving improved central, cardiovascular and endothelium coupling to maintain sufficient tissue oxygenation and reduce primary and secondary "hit" complications. As with any new drug innovation, translation into humans is challenging.
One of the most commonly used battlefield resuscitation fluid is HES, which there are increasing questions about its benefits. 7% saline/ALM may be a big improvement on this but there needs to be human studies.